disappears clinic gestosis

The earliest manifestation of human organnogo blood flow is proteinuriya. 4. Despite the progression of arterial and venous vazodilyatatsiya vazospazma persists. Relative overflow of venous vascular bed leads to a) the maintenance of a low venous return, arterial gipovolemii, vazospazma, b) increase the likelihood and extent swells. When expressed gipovolemii PE is not accompanied by perifericheskimi circulation. It is known that such "dry" PE is accompanied by a maximum risk of maternal and perinatal mortality. 5. Over the PE clinical manifestation in the second half of pregnancy associated with a maximum capacity of the vascular bed in the period : IPC, the hormone. It should be noted that the rule, and possibly in women with PE until the second half of pregnancy continuing boom RAAS (15), and with it the growth of BCC. 6. Relative gipovolemiya streamlined with the slowing down of the RAAS, depending on its degree can be : * with the early timing of pregnancy : before 16-18 weeks. The reduced amount of blood uterus resulting in the violation of invasion trofoblasta in muscle layer spiral arteries (in 8-10 weeks and 16-18 weeks) and the formation of abnormal IPC, which translates into high resistance krovotoku in uterine arteries (Ma). The increased risk of early manifestation of symmetrical shape VZRP and symptoms inadequate. * in the second half of pregnancy, when the invasion trofoblasta in spiral arteries completed. The IPC, according to the blood flow in dopplerometrii Ma remained within normal limits, and risk VZRP and inadequate substantially reduced. 7. with the early timing of pregnancy : before 16-18 weeks. The reduced amount of blood uterus resulting in the violation of invasion trofoblasta in muscle layer spiral arteries (in 8-10 weeks and 16-18 weeks) and the formation of abnormal IPC, which translates into high resistance krovotoku in uterine arteries (Ma). The increased risk of early manifestation of symmetrical shape VZRP and symptoms inadequate. 8. in the second half of pregnancy, when the invasion trofoblasta in spiral arteries completed. The IPC, according to the blood flow in dopplerometrii Ma remained within normal limits, and risk VZRP and inadequate substantially reduced. 9. After the births to women with PE chance hormone status and reduces vascular bed (the disappearance of the IPC, spadenie veins, obliteratsiya receptacles), which are close to or in line with the BCC. Disappears ishemizirovanny channel Body : Rokthaego-source sosudosuzhivayuschih incentives. As a result, recovering venous return and cardiac ejection allowed hypertension, recovering organny bloodstream, ie disappears clinic gestosis. 10. Virtually no PE in povtornorodyaschih seems related to feature enzymes, including enzymes RAAS, to increase their activity in the second "summit" with the substrate. However, with repeated pregnancies, initially low blood plasma leads to the development of hypertension (26). 11. Lack of PE in animals can be traced to function bipedal locomotion rights. In horizontal or bent torso animals even in the relative gipovolemii and venous vazodilyatatsii no significant reduction in venous return, as we have seen in humans, and less significant decrease cardiac ejection. It is also possible that the genome of animal parts, regulatory activity RAAS is significantly different from the human genome. Table 1 presents outcomes of pregnancy in women with normal levels of central hemodynamics (TSGD). Screening pregnant on TSGD very accurately identifies a group of women with favorable pregnancy outcomes for the mother and fetus. Pregnant with low TSGD required more in-depth survey, possibly in a hospital environment.