clinical disease

The formation of PS may participate as congenital or acquired factors. A limited number of studies to determine the real frequency (incidence), PS 135, 40J. Among the patients, surgery on the lumbar vertebrogennyh syndromes, patients with PS is currently range from 3 to 6% [2, 40]. Despite the fact that perceptions of clinical manifestations, diagnosis and treatment of PS emerged mainly over the past decade, the first message of peace to narrow pozvonochnom canal dates back to the beginning of the century. For the first time dekompressivnaya laminektomiya the lumbar level phenomena defeat roots horse tail was performed in 1900 [92], then in 1911 [13]. The cause of neurological violations considered ancylosing spondylitis, but operating findings included thickening spina bifida, increased steel joints and suffering yellow ligaments. Suinita M. (1910) first described the clinical picture of defeat roots horse tail in ahondroplazii, explaining the symptoms of congenital narrow spinal canal [98J. Elsberg S. (1913), and F. Kennedy with Moscow. (1914) presented clinical disease with unknown etiology, accompanied typical manifestations neyrogennoy peremezhayuscheysya leg characteristic of PS [32, 60]. Further understanding of the PS little changed by mid-century. C. Van Gelderen [105] described a patient with symptoms defeat lumbar roots, which will appear in walking and cutting regressiruyuschey in bringing those violations to the overdetermination yellow ligaments. Later V. Berginark [16] described the "transient spinalnuyu klaudikatsiyu" two patients, explaining the emergence of pain in the legs while walking defeat spinal roots. Since 1949 N. Veibiest published an extensive series of works on the database. idiopaticheskomu PS, which analyzed links anatomical changes spinal canal with a specific clinic defeat roots horse tail, defining it as neyrogennuyu "peremezhayuschuyusya hromotu (" claudicatio intermittens ") or psevdoklaudikatsiyu